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The July 2011 issue of the American Journal of Preventive Medicine reviews statistics on fatal TBIs from 2003-2008. During those years the percentage of fatal TBIs from motor vehicle accidents (MVAs) continued to decline while those from falls continued to rise. As of 2008 fatal TBIs from MVAs accounted for 31% of all
Brigid Waldron-Perrine, Ph.D., a recent graduate from Wayne State University, and her mentor, Lisa J. Rapport, Ph.D., professor of psychology at Wayne State University’s College of Liberal Arts and Sciences, reported in June 2011 that personal faith in a higher power improves the emotional and physical outcome of rehabilitation for TBI patients. Their
In July 2011 Dr. Theresa Pape announced that by using a series of TMS treatments she had significantly increased alertness in Josh Villa, a father of 3 children left in a deep coma after an auto accident 6 years before. TMS or transcranial magnetic stimulation uses a coil to send harmless magnetic pulses
While MRI can detect brain swelling/compression and CT can detect brain bleeding, neither type of scanner can find small traumatic brain lesions involving torn axons. So many mild and some moderate traumatic brain injuries remain invisible. However, a team of researchers at UC San Diego is making use of a new high tech
Soliders coming back from Iraq and Afghanistan with concussive brain injuries from explosions show problems with concentration, working memory, and multi-tasking. Dr. David Twillie, director of the Fort Campbell Traumatic Brain Injury Clinic, has developed a new method of treating these “mild” brain injuries with an 80% success rate. The method involves identifying
NeuroVive Pharmaceutical, AB is a Swedish drug development company. Working with NeuroStat it has developed a mechanism for delivering drugs directly to the brain through the blood-brain-barrier which normally screens out large molecules. In recent tests NeuroVive has shown that cyclosporine A, a drug used to help organ transplant recipients avoid immune rejection,
Some of the soldiers returning from Iraq and Afghanistan with TBI are displaying the dual problem of seizures and migraine. Two University of Utah researchers (K.C. Brennan, M.D., assistant professor of Neurology, and Edward Dudek, Ph.D., professor and chair of the Department of Physiology) are teaming up with the Department of Defense to
Benjamin Cravatt, Ph.D., at the Scripps Research Institute and Daniel Nomura, Ph.D., at UC Berkeley have made an important discovery about how to block brain inflammation, something which can severely compound the initial damage done by a TBI. They learned that in the brain the production of arachidonic acid (which gets converted into
Lithium has been used for decades to treat mania. Psychiatrists believe that lithium controls mania in part by decreasing the activity of the excitatory neurotransmitter glutamate. Following TBI some brain cells release excessive quantities of glutamate which damages or kills other brain cells. Fengshan Yu and colleagues at NIH and the University of
Steven Flanagan, M.D., Chairman of the Department of Rehabilitation Medicine at the NYU’s Rusk Medical Institute is an expert on rehabilitation of adult and child traumatic brain injuries. In December 2011 he will give a talk in Orlando, Florida, offering a rationale for treating TBI as a chronic disease with long-lasting medical problems
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